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Other strategies focus on preventing the major complication of HZ – post- herpetic neuralgia.

This crucial role of VZV-specific cell-mediated immunity suggests that boosting these responses by vaccination will be an effective strategy for reducing the burden of HZ. SUMMARY Impairment of varicella zoster virus (VZV)-specific cell-mediated immunity, including impairment due to immunosenescence, is associated with an increased risk of developing herpes zoster (HZ), whereas levels of anti-VZV antibodies do not correlate with HZ risk. Prevention strategies for herpes zoster and post- herpetic neuralgia Diagnosis by antibody testing led to aggressive antiviral treatment and a favorable outcome. Detection of anti-varicella zoster virus (VZV) IgM and VZV IgG antibody in cerebrospinal fluid (CSF), with reduced serum/CSF ratios of anti-VZV IgG compared to normal serum/CSF ratios for albumin and total IgG, proved that VZV caused the protracted neurological complications. Morita, Y Osaki, Y Doi, Y Forghani, B Gilden, D HĪfter lumbar-distribution zoster, an HTLV-1-seropositive woman developed chronic radicular sacral-distribution pain ( zoster sine herpete), cervical-distribution zoster paresis and thoracic-distribution myelopathy.

Chronic active VZV infection manifesting as zoster sine herpete, zoster paresis and myelopathy.